Corrigendum to “Graphitic-C₃N₄/chitosan-doped NiO nanostructure to treat the polluted water and their bactericidal with in silico molecular docking analysis” [Int. J. Biol. Macromol. 227 (2023) 962–973, (S014181302202832X), (10.1016/j.ijbiomac.2022.11.273)]

The authors regret Fig. 3(a) should be replaced with the provided figure. [Formula presented] The line “Upon doping of CS in NiO NPs, a distinctive peak at around 2θ of 13.5° is detected, demonstrating the presence of dopant [50]” should be read as “No distinctive peak of CS was observed in the CS-NiO spectrum due to the small amount of the dopant”. The line “Debye-Scherrer formula was used to calculate crystallite size, which was found to be 41.77 nm, 34.19 nm, 26.60 nm, 67.25 nm and 25.70 nm for dopant free and (2, 4%) g- C₃N₄/CS doped NiO respectively” should be read as “Debye-Scherrer formula was used to calculate crystallite size, which was found to be 67.25, 34.19, 41.77, and 26.60 nm for dopant free, CS-NiO and (2, 4 wt.%) of g-C₃N₄ doped CS-NiO, respectively”. The line “Similarly, DNA gyrase (PDB code: 6KZX) [45] and enoyl carrier acyl protein (Fab1, PDB code: 1MFP) [46] were retrieved from RCSB PDB (www.rcsb.org) and processed in sybyl” should be read as “Similarly, beta-Lactamse (PDB code: 1MWU) [45] and β-ketoacyl-acyl carrier protein synthase III (FabH, PDB code: 5BNM) [46] were retrieved from RCSB PDB (www.rcsb.org) and processed in sybyl.” References “[45] and [46]” should be replaced with provided references [45] Lim, D. and Strynadka, N.C., 2002. Structural basis for the β lactam resistance of PBP2a from methicillin-resistant Staphylococcus aureus. Nature Structural Biology, 9(11), pp.870–876. [46] McKinney, D.C., Eyermann, C.J., Gu, R.F., Hu, J., Kazmirski, S.L., Lahiri, S.D., McKenzie, A.R., Shapiro, A.B. and Breault, G., 2016. Antibacterial FabH inhibitors with mode of action validated in Haemophilus influenzae by in vitro resistance mutation mapping. ACS Infectious Diseases, 2(7), pp.456–464.The authors would like to apologise for any inconvenience caused.